Welcome to Kaanglab

Institute for Basic Science




1. Li XY, Ko HG, Chen T, Descalzi G, Koga K, Wang H, Kim SS, Shang Y, Kwak C, Park SW, Shim J, Lee K, Collingridge GL, Kaang BK, Zhuo M, 2010: Alleviating neuropathic pain hypersensitivity by inhibiting PKMzeta in the anterior cingulate cortex. Science, 330(6009),1400-4

2. Deok-Jin Jang, Jin-A Lee, Yeon-Su Chae, Bong-Kiun Kaang, 2010: State-dependent disruption of short-term facilitation due to overexpression of the apPDE4 supershort form in Aplysia. Molecules and Cells

3. Jang DJ, Park SW, Lee JA, Lee C, Chae YS, Park H, Kim MJ, Choi SL, Lee N, Kim H, Kaang BK, 2010: termini of apPDE4 isoforms are responsible for targeting the isoforms to different cellular membranes. Learn Mem, 17(9),469-79

4. Choi SL, Lee YS, Rim YS, Kim TH, Moroz LL, Kandel ER, Bhak J, Kaang BK, 2010: Differential Evolutionary Rates of Neuronal Transcriptome in Aplysia kurodai and Aplysia californica as a Tool for Gene Mining. J Neurogenet, 24(2),75-82

5. Lee HR, Kaang BK, 2010: A novel analytic measure for the water maze utilizing the concept of entropy. Front Neurosci, 4,1

6. Park JW, Park AY, Lee S, Yu NK, Lee SH, Kaang BK, 2010: Detection of TrkB Receptors Distributed in Cultured Hippocampal Neurons through Bioconjugation between Highly Luminescent (Quantum Dot-Neutravidin) and (Biotinylated Anti-TrkB Antibody) on Neurons by Combined Atomic Force Microscope and Confocal Laser Sca. Bioconjug Chem, 21(4),597-603

7. Park SW, Ko HG, Lee N, Lee HR, Rim YS, Kim H, Lee K, Kaang BK, 2010: Aged wild-type littermates and APPswe+PS1/ΔE9 mice present similar deficits in associative learning and spatial memory independent of amyloid load. Genes & Genomics, 32(1),63-70

8. Chae YS, Lee SH, Cheang YH, Lee N, Rim YS, Jang DJ, Kaang BK, 2010: Neuronal RNA granule contains ApCPEB1, a novel Aplysia CPEB, in Aplysia sensory neuron. Exp Mol Med, 42(1),30-7

9. Choi JH, Kim JE, Kaang BK, 2010: Protein synthesis and degradation are required for the incorporation of modified information into the pre-existing object-location memory. Mol Brain, 3,1